Anabolic Steroid • Oral • Schedule III

Anadrol: The Most Powerful Oral Mass Builder

Last updated: March 2026

Oxymetholone (Anadrol) is the most potent oral anabolic steroid available. FDA-approved for aplastic anemia and HIV-related muscle wasting, it produces rapid, dramatic mass gains — paired with significant hepatotoxicity and water retention that demands careful monitoring and harm-reduction protocols.

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Typical Daily Dose
Clinical & Community Data
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Anabolic Rating
(vs. Testosterone = 100)
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Oral Bioavailability
Mechanism

How Anadrol Works

Oxymetholone's unique pharmacology sets it apart from other oral steroids. It activates androgen receptors directly and appears to activate estrogen receptors independently of aromatization — a mechanism not fully elucidated in the literature.

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17α-Alkylation — Oral Survival

The 17α-methyl group prevents first-pass hepatic breakdown, allowing oral bioavailability. This same modification is responsible for Anadrol's severe hepatotoxicity. ALT and AST enzymes typically rise 3-5× baseline within weeks of use. This is a direct chemical consequence of the modification, not reversible through timing or dose.

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Estrogen Receptor Activation — No Aromatase Needed

Unlike most androgens, oxymetholone does not aromatize to estradiol, yet produces profound estrogenic effects: water retention, bloating, gynecomastia risk. Research suggests oxymetholone acts as a partial agonist at the estrogen receptor directly. Standard AIs may not fully suppress these effects since serum E2 may remain normal.

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Erythropoiesis — FDA-Approved Mechanism

Anadrol's clinical approval rests on its ability to stimulate erythropoiesis (red blood cell production). It increases EPO sensitivity, raises hematocrit and hemoglobin, and improves oxygen delivery. This is why it remains FDA-approved for aplastic anemia, Fanconi anemia, and HIV-associated wasting. Hematocrit monitoring is essential — elevated levels raise stroke risk.

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Nitrogen Retention & Protein Synthesis

Oxymetholone dramatically improves nitrogen retention and upregulates protein synthesis pathways. Clinical studies in HIV patients show 8kg lean mass gains over 16 weeks at therapeutic doses. Community-reported gains of 5-10kg in 4-6 week cycles are consistent with this data, though most gains include substantial water (which reverses post-cycle).

What the Evidence Shows

Clinical data from HIV-wasting and anemia trials, supplemented with community-reported outcomes (labeled).

Lean Mass Gain (HIV Wasting, 100mg/day, 16 weeks)
Hengge et al. 1996 — clinical trial data
+8kg mean
Hemoglobin Increase
Aplastic anemia trials — erythropoietic effect
~30-50%
ALT/AST Elevation
Liver enzyme increase — hepatotoxicity signal
3-5× ULN
Community-Reported Water Retention
Anecdotal — most common complaint in community surveys
~85% report
Strength Increase (Community)
Anecdotal — powerlifters report fastest strength gains of any oral
Fastest oral

Risks & Side Effects

Hepatotoxicity
Severe — peliosis hepatis, cholestatic hepatitis, hepatocellular carcinoma risk with chronic use
Very High
Cardiovascular Strain
LDL increase, HDL suppression, hematocrit elevation
High
HPTA Suppression
Endogenous testosterone shutdown — requires PCT
Complete
Water Retention & Bloating
Due to direct estrogen receptor activation
Severe
Androgenic Effects
Acne, hair loss, virilization (women)
Moderate-High

Key Takeaways

✅ What We Know
  • FDA-approved for aplastic anemia and HIV wasting since 1961
  • Most powerful oral AAS by anabolic rating (320 vs testosterone's 100)
  • Does NOT aromatize, but still causes estrogenic effects via direct ER activation
  • Rapid mass and strength gains in 4-6 week cycles
  • TUDCA and NAC are the primary liver-support supplements used alongside it
  • All gains include significant water weight — expect substantial post-cycle loss
🚨 Critical Risks
  • Among the most hepatotoxic substances studied — never use without liver bloodwork
  • Cycle length must be limited (4-6 weeks max in community practice)
  • Contraindicated with other hepatotoxic drugs or alcohol
  • Peliosis hepatis (blood-filled liver cysts) is a rare but life-threatening risk
  • Polycythemia (high RBC) raises stroke and thrombosis risk — monitor hematocrit

🛒 Liver Support & Monitoring

If researching Anadrol, liver protection and bloodwork monitoring are non-negotiable harm-reduction practices.

Related Resources

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⚠️ Legal & Medical Disclaimer

This page is for educational and harm-reduction purposes only. Oxymetholone (Anadrol) is a Schedule III controlled substance under the Anabolic Steroid Control Act. Possession without a valid prescription is illegal in the United States and many other countries. This content does not constitute medical advice and is not a recommendation to use this substance. Always consult a licensed physician before using any anabolic agent. Misuse of anabolic steroids carries serious legal and health consequences.