Esterified SARM • Oral • Research

RAD-150 (TLB-150): The Long-Acting RAD-140 Analog

Last updated: March 2026

RAD-150 (TLB-150) is an esterified analog of RAD-140 (Testolone), featuring a benzoate ester that extends half-life and improves plasma stability. It acts as an androgen receptor agonist with anabolic selectivity. A newer research chemical with extremely limited human data — all findings come from preclinical animal models.

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Typical Research Dose
Once daily oral
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Estimated Half-Life
Extended by ester
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Ester Type
Cleaved to RAD-140 in vivo

How RAD-150 Works

RAD-150 is a prodrug of RAD-140. The benzoate ester attached to the parent molecule is hydrolyzed by endogenous esterases in the bloodstream, releasing active RAD-140. The result is the same core androgen receptor agonism as RAD-140, delivered with a longer, smoother pharmacokinetic profile.

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Ester Hydrolysis Prodrug

RAD-150 carries a benzoate ester group attached to the RAD-140 base structure. After oral ingestion, plasma esterases cleave this ester bond, releasing free RAD-140 into circulation. This prodrug mechanism is the same strategy used with testosterone esters (enanthate, cypionate) to extend release kinetics without altering the active molecule's pharmacology.

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Androgen Receptor Full Agonism

The released RAD-140 binds the androgen receptor (AR) as a full agonist with high affinity. Unlike partial agonists (e.g., Andarine), RAD-140 fully activates AR-dependent transcription in target tissues. Animal studies of RAD-140 show a high anabolic-to-androgenic ratio — strong effects in muscle and bone with comparatively reduced prostate and scalp stimulation.

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Extended Plasma Stability

RAD-140's reported half-life is ~15-20 hours. The benzoate ester in RAD-150 is estimated to extend this to ~48 hours, though no peer-reviewed pharmacokinetic data in humans exists. The slower ester hydrolysis provides a more stable plasma concentration curve, theoretically reducing peak-trough fluctuations that may contribute to side effects or inconsistent tissue exposure.

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Anabolic Gene Transcription

AR agonism in skeletal muscle activates anabolic gene programs: increased protein synthesis, IGF-1 expression, satellite cell recruitment, and anti-catabolic signaling. RAD-140 animal data demonstrated significant lean mass preservation even in castrated rodent models, suggesting high tissue-selective potency. RAD-150 is presumed to share this profile once the ester is cleaved.

What Preclinical Studies Show

⚠️ All data below is from animal studies (primarily rodent and primate models of RAD-140, the parent compound). No peer-reviewed human clinical data exists for RAD-150 specifically. These figures are for research reference only.

Lean Mass Preservation vs Testosterone (castrated rat model)
RAD-140 parent compound — animal study
~90%
Prostate Weight (androgenic effect vs testosterone)
RAD-140 anabolic:androgenic selectivity — animal study
~4%
Neuroprotective Effect — Brain Cell Survival (kainate model)
RAD-140 in vitro and rat model — animal/cell study
Significant
Estimated Half-Life Extension vs RAD-140
Projected from ester chemistry — no published PK data
~2–3×
Testosterone Suppression (HPG axis)
Expected based on AR agonism mechanism — class effect
Moderate–High
Bone Mineral Density Retention
RAD-140 in osteoporosis rodent model — animal study
Positive

Side Effects & Risks

Testosterone / HPG Axis Suppression
Expected with full AR agonism — bloodwork monitoring essential
Moderate–High
Liver Enzyme Elevation (ALT/AST)
Case reports associated with RAD-140; RAD-150 data absent
Low–Moderate
Androgenic Effects (acne, hair loss)
Low androgenic ratio in animal models — human data absent
Low (estimated)
Mood / Aggression Changes
Reported anecdotally with RAD-140; mechanism unclear
Low–Moderate
Unknown Ester-Specific Risks
Novel ester — benzoate metabolism in humans not characterized
Unknown

Key Takeaways

✅ What We Know
  • Esterified prodrug of RAD-140 — benzoate ester cleaved to active compound in vivo
  • Parent RAD-140 has strong animal-model evidence for anabolic selectivity
  • High anabolic:androgenic ratio in rodent studies (~90% anabolic, ~4% androgenic vs testosterone)
  • Once-daily dosing is theoretically sufficient given extended estimated half-life
  • Expect HPG axis suppression and testosterone reduction — monitor bloodwork
  • RAD-140 has documented liver enzyme elevation in human case reports
⚠️ What We Don't Know
  • No peer-reviewed human pharmacokinetic data for RAD-150 exists
  • True half-life in humans is uncharacterized — ~48hr is a projection
  • Benzoate ester metabolism and any unique risks are unknown
  • Long-term safety, carcinogenicity, and reproductive effects not studied

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⚠️ Important Disclaimer

This page is for educational purposes only. It is not medical advice. RAD-150 (TLB-150) is not FDA approved and is not intended for human use. All data referenced is from preclinical animal studies of the parent compound RAD-140 or theoretical ester pharmacokinetics. Do not use any research chemical without consulting a qualified medical professional.